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Canada-0-FreightForwarding ไดเรกทอรีที่ บริษัท
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ข่าว บริษัท :
- Long-term cadmium exposure impairs cognitive function by activating lnc-
Cadmium (Cd), a ubiquitous environmental contaminant, is deemed a possible aetiological cause of cognitive disorders in humans Nevertheless, the exact mechanism by which chronic exposure to Cd causes neurotoxicity is not fully understood
- Cadmium Exposure Impairs Adult Hippocampal Neurogenesis
Cadmium (Cd) is an environmental pollutant of considerable interest throughout the world and potentially a neurotoxicant Our recent data indicate that Cd exposure induces impairment of hippocampus-dependent learning and memory in mice
- Cadmium Exposure Impairs Adult Hippocampal Neurogenesis
To our knowledge, these findings provide some of the first direct evidence that Cd exposure impairs adult hippocampal neurogenesis, which may contribute to Cd-induced deficits in hippocampus-dependent learning and memory
- Cadmium‐induced global DNA hypermethylation promoting mitochondrial . . .
This study used hippocampal neurons cultured in vitro to observe the impact of cadmium exposure on mitochondrial dynamics and apoptosis
- Cadmium and Its Neurotoxic Effects | Springer Nature Link
This understanding sets the stage for a detailed exploration of the neurological effects of cadmium It highlights cadmium-induced neurotoxicity, its manifestations, and the impacts on the central nervous system (CNS)
- Cadmium‐induced global DNA hypermethylation promoting mitochondrial . . .
This study shows that cadmium affects hippocampal neurons by enhancing genomic methylation levels, changing mitochondrial characteristics, and promoting apoptosis, revealing a new mechanism of cadmium - induced neurotoxicity
- Cadmium-induced gut dysbiosis precedes the onset of hippocampus . . .
We found that Cd exposure caused gut dysbiosis before the onset of cognitive deficits, with specific bacterial species correlating with impaired cognition RNA sequencing revealed alterations in the expression of genes involved in cognition and neuroinflammation in the hippocampus
- Mechanisms of Cadmium Neurotoxicity - PMC
Cadmium gains entry into the nervous system via zinc and calcium transporters, altering the homeostasis for these metal ions Once within the nervous system, cadmium disrupts mitochondrial respiration by decreasing ATP synthesis and increasing the production of reactive oxygen species
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